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- 13D markerless asymmetry analysis
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- 1Adolescent Idiopathic Scoliosis (AIS)
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Algorithms for Adolescent Idiopathic Scoliosis Classification Based on Surface Topography Analysis
DownloadSpring 2018
While the common method for diagnosing and monitoring adolescents with idiopathic scoliosis (AIS) is X-ray radiographs from which a Cobb angle is measured, studies have shown that high radiation exposure is linked to high risk of cancer, particularly, for children and women. This thesis describes...
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Markerless 3D Assessment of Severity and Progression of Scoliosis Using Surface Topography on Isolated Back Scans
DownloadSpring 2019
The objectives of the thesis were to: 1. Determine if open source software can be use in markerless asymmetry analysis of surface topography. 2. Determine if isolated back scans can be used in markerless asymmetry analysis of surface topography. Markerless asymmetry analysis of surface...
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A Novel 3D Printed Brace Manufacturing Process for the Treatment of Adolescent Idiopathic Scoliosis
DownloadSpring 2019
prototype brace wear with donning and doffing was conducted. The FDM, Nylon12, 2.5 mm thick, and upright printed prototype brace showed retention of structural integrity with 2 years brace wear simulation. To understand the pressures applied by orthotists to obtain the satisfactory simulated in-brace body
the traditional brace. However, it is 27% more expensive in cost because the 3D printed brace was printed from external source while the traditional brace was built in-house. In conclusion, this thesis reports a new design of dynamic brace pads which were implemented into a novel 3D printed brace
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Investigating the Relationship Between LUNATIC FRINGE Variants and Spondylocostal Dysostosis Type-III
DownloadFall 2024
estimated to be as high as 1-in-33, and treatment ranges from physiotherapy and structural bracing to corrective surgery. In most cases, early diagnosis and proactive care are key to limiting curve progression and minimizing treatment invasiveness. However, the underlying cause of most scoliosis cases is
and/or enzymatically dysfunctional. Finally, the phenotype of the axial skeleton, but not elsewhere, may be modulated in a variant-specific fashion, supporting our previous work. More reports are needed to continue testing this hypothesis. We anticipate our data will be used as a basis for the